The role of reactive oxygen species and epigenetics in the fetal programming of hypertension by glucocorticoids
dc.contributor.author | Lamothe, Jeremy D. | |
dc.date.accessioned | 2021-01-20T20:36:37Z | |
dc.date.available | 2021-01-20T20:36:37Z | |
dc.date.issued | 2020-08-20 | |
dc.description.degree | Philosophy (PhD) in Biomolecular Sciences | en_US |
dc.identifier.uri | https://laurentian.scholaris.ca/handle/10219/3623 | |
dc.language.iso | en | en_US |
dc.publisher.grantor | Laurentian University of Sudbury | en_US |
dc.subject | epigenetics | en_US |
dc.subject | reactive oxygen species | en_US |
dc.subject | fetal programming | en_US |
dc.subject | hypertension | en_US |
dc.subject | glucocorticoids | en_US |
dc.subject | DNA methyltransferase | en_US |
dc.subject | Histone deacetylase | en_US |
dc.subject | HPA | en_US |
dc.subject | Catecholamines | en_US |
dc.title | The role of reactive oxygen species and epigenetics in the fetal programming of hypertension by glucocorticoids | en_US |
dc.type | Thesis | en_US |